主讲人:Professor Sebastian Schreiber,University of California, Davis
时间:2019年6月19日14:30
地点:3号楼332报告厅
举办单位:数理学院
内容介绍:A long standing, fundamental question in biology is what are the minimal conditions to ensure the long-term persistence of a population, or to ensure the long-term coexistence of interacting species? The answers to this question are essential for identifying mechanisms that maintain biodiversity and guiding conservation efforts. Mathematical models play an important role in identifying potential mechanisms and, when coupled with empirical work, can determine whether or not a given mechanism is operating in a specific population or community. For over a century, nonlinear difference and differential equations have been used to identify mechanisms for population persistence and species coexistence. These models, however, fail to account for intrinsic and extrinsic random fluctuations experienced by all populations. In this talk, I discuss recent mathematical results about persistence and coexistence for models accounting for demographic and environmental stochasticity. Demographic stochasticity stems from populations consisting of a finite number of interacting individuals. These dynamics can be represented by Markov chains on a countable state space. For closed populations in a bounded world, extinction in these models occurs in finite time, but may be preceded by long-term transients. Quasi-stationary distributions (QSDs) of these Markov chains characterize this meta-stable behavior. These QSDs correspond to an eigenvector of the transition operator restricted to non-extinction states, and the associated eigenvalue determines the mean time to extinction when the Markov chain is in the quasi-stationary state. I will discuss under what conditions (i) this mean time to extinction increases exponentially with habitat size and (ii) the QSDs concentrate on attractors of the mean field model of the Markov chain. These results will be illustrated with models of competing California annual plant species, and a classical model of host-pathogen interactions.